Anatomy
3 questionsA person is not able to extend his metacarpophalangeal joint. Injury to which of the following nerve result in this?
Which of the following statements is true regarding the anatomy of the external nose?
Which of the following arteries does NOT contribute to Little's area?
NEET-PG 2013 - Anatomy NEET-PG Practice Questions and MCQs
Question 991: A person is not able to extend his metacarpophalangeal joint. Injury to which of the following nerve result in this?
- A. Posterior Interosseous nerve injury (Correct Answer)
- B. Radial nerve injury
- C. Ulnar nerve injury
- D. Median nerve injury
Explanation: Posterior Interosseous Nerve (PIN) injury - The Posterior Interosseous Nerve is the deep motor branch of the radial nerve that specifically innervates the extensor muscles of the fingers and thumb - These muscles include: Extensor Digitorum, Extensor Indicis, Extensor Digiti Minimi, Extensor Pollicis Longus and Brevis [1] - PIN injury causes inability to extend the MCP joints and interphalangeal joints of the fingers [1] - Wrist extension is preserved because the Extensor Carpi Radialis Longus (ECRL) and often ECRB are innervated by the radial nerve proper before it gives off the PIN [1] - This results in a characteristic finger drop without wrist drop Radial nerve injury - A high radial nerve injury (proximal, above the elbow) would cause both wrist drop AND finger extension loss - However, radial nerve injury at the spiral groove (most common site) typically spares the PIN or affects it less severely - The question asks specifically about isolated inability to extend MCP joints, which is the hallmark of PIN injury, not general radial nerve injury - Radial nerve proper gives branches to triceps, brachioradialis, and ECRL before dividing into PIN and superficial branch Ulnar nerve injury - The ulnar nerve innervates intrinsic hand muscles (interossei, lumbricals to digits 4-5, hypothenar muscles, adductor pollicis) [1] - Ulnar nerve injury causes claw hand deformity with MCP hyperextension (not loss of extension) and IP joint flexion - This is the opposite of what is described in the question Median nerve injury - The median nerve innervates the thenar muscles, lateral two lumbricals, and forearm flexors [1] - Median nerve injury causes ape hand deformity with loss of thumb opposition and flexion - It does not affect MCP joint extension, which is an extensor function
Question 992: Which of the following statements is true regarding the anatomy of the external nose?
- A. The lower one-third is primarily cartilaginous. (Correct Answer)
- B. The upper two-thirds is entirely bony.
- C. The lateral aspect has only a single cartilage.
- D. The external nose is supported by two nasal bones.
Explanation: ***The lower one-third is primarily cartilaginous.*** - The **lower one-third** of the external nose, including the nasal tip and alae, is predominantly supported by **alar cartilages** (lower lateral cartilages) and other minor cartilages, giving it flexibility. - This cartilaginous structure allows for movement and shaping of the nostrils. - This statement is **anatomically accurate and complete**. *The upper two-thirds is entirely bony.* - This is **incorrect**. - The **upper one-third** is bony (nasal bones and frontal process of maxilla). - The **middle one-third** is primarily **cartilaginous** (upper lateral cartilages). - Therefore, the upper two-thirds consists of **both bone and cartilage**, not entirely bone. *The lateral aspect has only a single cartilage.* - This is **incorrect**. - The lateral aspect contains **multiple cartilages**: upper lateral cartilages, lower lateral (alar) cartilages, and accessory cartilages. - The presence of multiple cartilages provides structural support and flexibility. *The external nose is supported by two nasal bones.* - This is **incomplete and misleading**. - While two **nasal bones** do form the superior bony bridge (upper one-third), the external nose is also supported by: - Frontal process of the maxilla - Upper and lower lateral cartilages - Septal cartilage - Stating only the nasal bones ignores the majority of nasal support structures.
Question 993: Which of the following arteries does NOT contribute to Little's area?
- A. Sphenopalatine artery
- B. Posterior Ethmoidal artery (Correct Answer)
- C. Greater palatine artery
- D. Anterior Ethmoidal artery
Explanation: Posterior Ethmoidal artery - The posterior ethmoidal artery primarily supplies the posterior ethmoidal cells and part of the sphenoid sinus, but it does not contribute to the vascular plexus in Little's area. - Little's area, also known as Kiesselbach's plexus, is formed by anastomoses of several arteries on the anterior nasal septum. Sphenopalatine artery - The sphenopalatine artery, a terminal branch of the maxillary artery, is a major contributor to Little's area through its septal branch. - It supplies a significant portion of the nasal septum and is frequently involved in posterior epistaxis. Greater palatine artery - The greater palatine artery, a branch of the descending palatine artery (from the maxillary artery), enters the nasal cavity through the incisive canal and contributes to Little's area on the nasal septum. - It primarily supplies the hard palate and then anastomoses with other vessels in the anterior nasal septum. Anterior Ethmoidal artery - The anterior ethmoidal artery, a branch of the ophthalmic artery, is a key contributor to Little's area. - It supplies the anterior and middle ethmoidal cells and also contributes to the blood supply of the dura mater.
ENT
1 questionsWhat is not true about the use of intranasal steroids in nasal polyposis?
NEET-PG 2013 - ENT NEET-PG Practice Questions and MCQs
Question 991: What is not true about the use of intranasal steroids in nasal polyposis?
- A. Effective in all types of nasal polyps (Correct Answer)
- B. May cause nasal irritation
- C. Reduce recurrence
- D. Most effective in eosinophilically predominant polyps
Explanation: ***Effective in all types of nasal polyps*** - Intranasal steroids are primarily effective in nasal polyps with an **eosinophilic inflammatory component**, which is the most common type. - They are **not effective in all types** - efficacy is significantly reduced in polyps with **neutrophilic inflammation** or those related to conditions like **cystic fibrosis**, reflecting different underlying pathologies. - This statement is **FALSE**, making it the correct answer to this negation question. *May cause nasal irritation* - **Nasal irritation**, including **burning, stinging**, or **dryness**, is a common local side effect associated with the use of intranasal steroids. - Other local side effects can include **epistaxis** (nosebleeds) and mucosal atrophy, though less common. - This statement is **TRUE**. *Reduce recurrence* - **Intranasal steroids** are crucial in **reducing the recurrence** of nasal polyps after surgical removal. - Their anti-inflammatory action helps to **control the underlying inflammation** that contributes to polyp formation. - This statement is **TRUE**. *Most effective in eosinophilically predominant polyps* - Intranasal steroids primarily target the **eosinophilic inflammatory pathway**, which is characteristic of the majority of **chronic rhinosinusitis with nasal polyps (CRSwNP)**. - While they have **maximal efficacy** in eosinophilic polyps, they may have limited benefit in mixed inflammatory patterns. - Their efficacy is significantly reduced in polyps that are predominantly **neutrophilic** or associated with systemic conditions like **cystic fibrosis**, as these involve different inflammatory mechanisms. - This statement is **TRUE**.
Orthopaedics
5 questionsBlock vertebrae are seen in which condition?
What is a Hangman's fracture?
Most common cause of kyphotic deformity ?
Saturday night palsy is which type of nerve injury?
Which of the following movements is typically restricted in Perthes disease?
NEET-PG 2013 - Orthopaedics NEET-PG Practice Questions and MCQs
Question 991: Block vertebrae are seen in which condition?
- A. Pagets disease
- B. Leukemia
- C. TB
- D. Klippel-Feil syndrome (Correct Answer)
Explanation: ***Klippel-Feil syndrome*** - **Block vertebrae** are a characteristic radiographic finding in **Klippel-Feil syndrome**, resulting from the congenital fusion of two or more cervical vertebrae. - This fusion leads to a **short neck**, **low hairline**, and **restricted neck motion**. *Pagets disease* - **Paget's disease** is a chronic condition of abnormal bone remodeling, leading to enlarged and weakened bones. - While it can cause vertebral body changes, **block vertebrae** resulting from congenital fusion are not a typical feature. *Leukemia* - **Leukemia** involves uncontrolled proliferation of abnormal white blood cells, which can infiltrate bone marrow and cause lytic or blastic lesions in bones. - It does not cause **block vertebrae**, which are a developmental anomaly. *TB* - **Tuberculosis (TB) of the spine (Pott's disease)** is an infectious condition causing vertebral destruction, collapse, and kyphosis. - While TB can lead to vertebral collapse and eventual fusion during healing, the primary pathology is destructive and not the congenital fusion seen as **block vertebrae**.
Question 992: What is a Hangman's fracture?
- A. Fracture dislocation of C2 (Correct Answer)
- B. Fracture dislocation of ankle joint
- C. Fracture of odontoid
- D. Subluxation of C5 over C6
Explanation: ***Fracture dislocation of C2*** - A Hangman's fracture classically refers to a **bilateral fracture of the pars interarticularis of the axis (C2)**, often with an associated anterior subluxation of C2 on C3. - This injury is typically caused by **hyperextension-distraction forces**, such as those experienced in judicial hangings or motor vehicle accidents. *Subluxation of C5 over C6* - While cervical subluxations are serious, a **C5-C6 subluxation** does not specifically describe a Hangman's fracture. - This type of injury involves different vertebral levels and typically results from different mechanisms. *Fracture dislocation of ankle joint* - This option refers to an injury in the **lower limb**, completely unrelated to the cervical spine. - A Hangman's fracture is a specific type of **cervical vertebral fracture**. *Fracture of odontoid* - A fracture of the odontoid process involves the **dens (odontoid process)** of C2. - This is a distinct type of C2 fracture from a Hangman's fracture, which involves the **pars interarticularis**.
Question 993: Most common cause of kyphotic deformity ?
- A. Trauma
- B. Osteoporosis (Correct Answer)
- C. Ankylosing spondylitis
- D. Rickets
Explanation: ***Osteoporosis*** - **Osteoporosis** leads to vertebral compression fractures, particularly in the thoracic spine, which causes a gradual collapse of the vertebral bodies and an increase in the kyphotic curve. - This condition is very common, especially in **postmenopausal women** and the elderly, making it the most frequent cause of kyphotic deformity. *Trauma* - While significant **spinal trauma** can lead to kyphotic deformities, it is generally less common than the gradual kyphosis resulting from osteoporosis. - Traumatic kyphosis usually results from severe injuries leading to **vertebral body collapse** or neurological deficits. *Ankylosing spondylitis* - **Ankylosing spondylitis** can cause severe kyphosis, often referred to as a "bamboo spine," due to chronic inflammation and fusion of the vertebrae. - However, it is a less prevalent condition compared to **osteoporosis-related kyphosis**. *Rickets* - **Rickets**, a childhood bone disorder caused by **vitamin D deficiency**, can lead to bone deformities including kyphosis due to softened bones. - While a cause in children, its prevalence is lower than osteoporosis globally as a cause of kyphosis and it primarily affects a different age group.
Question 994: Saturday night palsy is which type of nerve injury?
- A. Neuropraxia (Correct Answer)
- B. Axonotemesis
- C. Complete section
- D. Neurotmesis
Explanation: ***Neuropraxia*** - This is the mildest form of nerve injury, involving a **temporary conduction block** without axonal disruption, often due to **compression** or mild stretching. - **Saturday night palsy**, caused by prolonged compression of the radial nerve, is a classic example, characterized by rapid and complete recovery, typically within days to weeks. *Axonotemesis* - This involves **axon damage** and Wallerian degeneration distal to the injury, but the **endoneurium and connective tissue sheaths remain intact**. - Recovery is slower and often incomplete, as it requires axonal regeneration through the preserved connective tissue tubes, taking months. *Neurotmesis* - This is the most severe type of nerve injury, involving **complete transection of the nerve fiber**, including the axon, myelin, and all connective tissue sheaths. - Recovery is often poor and requires surgical intervention to attempt re-approximation of the nerve ends. *Complete section* - This term is largely synonymous with **neurotmesis**, indicating a full anatomical disruption of the nerve. - It involves the severance of all nerve components, leading to complete loss of function distal to the injury and the poorest prognosis for spontaneous recovery.
Question 995: Which of the following movements is typically restricted in Perthes disease?
- A. Abduction & internal rotation (Correct Answer)
- B. Abduction & external rotation
- C. Adduction & internal rotation
- D. Adduction & external rotation
Explanation: ***Abduction & internal rotation*** - **Perthes disease** affects the femoral head, leading to pain and stiffness that most commonly restricts **abduction** and **internal rotation** of the hip. - This restriction is an early and consistent clinical finding, often accompanied by a ** Trendelenburg gait** due to gluteal muscle weakness or pain avoidance. *Abduction & external rotation* - While abduction can be restricted, a primary restriction in **external rotation** is less typical in early Perthes disease. - Reduced external rotation is more characteristic of conditions like **slipped capital femoral epiphysis (SCFE)**, especially in older children. *Adduction & internal rotation* - **Adduction** is generally preserved or even increased in Perthes disease as the hip seeks a position of comfort due to pain, making it an unlikely primary restriction. - While internal rotation is restricted, the combination with adduction restriction is not the classical presentation. *Adduction & external rotation* - Neither **adduction** nor **external rotation** are typically the primary hip movements restricted in Perthes disease. - Restriction in adduction is rare, and external rotation is often compensatory or less affected than internal rotation.
Pathology
1 questionsWhat are the common causes of vertebra plana?
NEET-PG 2013 - Pathology NEET-PG Practice Questions and MCQs
Question 991: What are the common causes of vertebra plana?
- A. Eosinophilic granuloma
- B. Metastatic disease
- C. Tuberculosis
- D. All of the options (Correct Answer)
Explanation: ***All of the options*** - **Vertebra plana** refers to the severe flattening of a vertebral body, often caused by a destructive lesion, and all listed options are known causes. [5] - While **eosinophilic granuloma** is a classic cause, **metastatic disease** and **tuberculosis** can also lead to significant vertebral collapse. [3], [5] *Eosinophilic granuloma* - This is a benign condition, a form of **Langerhans cell histiocytosis**, which commonly affects children and can cause vertebra plana. [1] - It results from proliferative lesions of **Langerhans cells** that infiltrate the bone, leading to its destruction and collapse. [1] *Metastatic disease* - **Malignant tumors** often spread to the spine, causing osteolytic lesions that weaken the vertebral body. [4], [5] - The destruction caused by metastatic deposits can lead to **vertebral collapse** and associated pain. [5] *Tuberculosis* - **Tuberculosis of the spine (Pott's disease)** is an infectious inflammatory condition that can severely damage vertebral bodies. [2], [3] - The granulomatous inflammation and caseous necrosis can erode the bone, leading to **vertebral collapse** and kyphosis. [2] **References:** [1] Kumar V, Abbas AK, et al.. Robbins and Cotran Pathologic Basis of Disease. 9th ed. Diseases of White Blood Cells, Lymph Nodes, Spleen, and Thymus, p. 630. [2] Kumar V, Abbas AK, et al.. Robbins and Cotran Pathologic Basis of Disease. 9th ed. Bones, Joints, and Soft Tissue Tumors, pp. 1197-1198. [3] Cross SS. Underwood's Pathology: A Clinical Approach. 6th ed. Common Clinical Problems From Osteoarticular And Connective Tissue Disease, pp. 669-670. [4] Kumar V, Abbas AK, et al.. Robbins and Cotran Pathologic Basis of Disease. 9th ed. The Lung, pp. 724-725. [5] Cross SS. Underwood's Pathology: A Clinical Approach. 6th ed. Common Clinical Problems From Osteoarticular And Connective Tissue Disease, pp. 671-672.